Dad’s Favorite French Bread (which we all know he uses for everything—including cinnamon rolls and scones) 2 1/4 c water (115 degrees) 2 Tbsp sugar 2 pkg (1 Tbsp each) active dry yeast 1 1/2 tsp salt 2 Tbsp cooking oil 6 c flour, stirred and measured In large mixing bowl, combine warm water and sugar. Sprinkle yeast over top; allow to soften. Add salt, cooking oil, and 3 c flour; beat well. Add remaining flour, stirring well with heavy spoon until dough gets too stiff. Then knead well with hands. Allow dough to rest 10 min. Knead down and allow to rest 10 min again. Knead. Repeat this process until dough has been kneaded five times. Turn dough out onto floured board. Knead two or three times to coat dough with light tough of four so it can be handled. Divide into two parts. Roll each part of dough into rectangle 9X12-inches. Roll dough up, starting from long side; pinch edge of loaf to seal. Arrange seam side down on large baking sheet that has been sprinkled with cornmeal or light sift of flour. Allow room for both loaves on the baking sheet. Repeat with the second part of dough. Cover lightly; allow to rise for 30 min. With very sharp knife, cut three gashes at an angle in top of each loaf; brush entire surface with egg wash (1 egg beaten slightly with 1 Tbsp water). If desired, sprinkle with sesame or poppy seeds. Bake at 400 for 30 min or until brown. Cool on racks. Makes 2 loaves. Note: For crustier loaf, a pan of hot water may be set on bottom of oven while bread is baking.
Melanie wrote: Still waiting for the results. The tests were done so late in the afternoon, that it will be Monday afternoon before my doctor is given the results and it might be Tuesday morning before she calls me. I still am having double vision, but only the left eye is affected by the palsy. If it is nerve damage, it should repair itself--maybe taking six months to fully heal. The numbness and other problems are unknowns at this time. I gave seven vials of blood and they will help the doctor to rule out a number of things such as Lyme disease, Rheumatoid arthritis, vitamin B12 deficiency, proteins, other wild antibodies, etc. So...waiting is the name of the game for now. If these tests are inconclusive, there will be a spinal tap in the next few weeks. Love you all. Thanks for your prayers. Melanie
Melanie will need more tests and is scheduled for more on Friday at 3 p.m. She may not have the answers to her problem right away but the nurologist she saw thought she may have something known as "Sixth Nerve Palsy." She did hit her head on the swing set while she was here. She didn't hit it hard enough to cause a bruise but it may have been just enough to cause the problem. I hade never heard of Sixth Nerve Palsy before. It is also referred to as Cranial Nerve Palsy or Abducens Nerve Palsy If an eye fails to move properly in all directions of gaze, a nerve palsy may be present, especially if the condition presents spontaneously in adulthood. Sixth Cranial Nerve Palsy presents with horizontal double vision, that is, the two images are horizontally misaligned. The double vision resolves when one eye is closed. In adults, the cause is usually a vascular infarct (diminished blood flow) of the nerve secondary to underlying diabetes or high blood pressure. Head trauma may also result in sixth cranial nerve palsy. In children, the condition usually follows a viral syndrome, though more serious intracranial inflammatory conditions and tumors must be considered. The diagnosis is usually easily confirmed by an ophthalmologist after observation of the eye movements in all fields of gaze. The affected eye will be unable to abduct (turn outwards beyond the midline). In adults with diabetes or high blood pressure and the sixth nerve palsy is the only other abnormal finding, a CT scan is usually not necessary. If there are any other concomitant neurological findings, pain, or a history of cancer, however, a CT scan will usually be obtained. In children, a CT scan is usually obtained to rule-out intracranial pathology. The prognosis for a full recovery in adults with diabetes or high blood pressure is good. However, recovery usually takes 3 to 6 months. Adults may elect to patch the eye to avoid double vision. In many cases, however, a temporary prism applied to the glasses may help restore single vision. The prism power may need changing every few weeks as the condition improves. For both children and adults in whom the condition fails to resolve, strabismus surgery may be considered.
Myrna wrote: LHT pointed out this morning that I had a typo in my writing. I typed the word "mine" instead of "mind" in the second to last sentence of the following paragraph. Please fix it before printing, or after, with a pen. Thanks, Mom. Delora Nebekker had to sell her home and move in with her son. She can no longer take care of herself. I work with her sister, Leta Jensen, at the temple and she said Delora was sad that she had to leave. I want to keep my mind as I age but I guess not knowing anything could, at times, be a blessing. Delora kept her mind. We have had a lot of changes in our ward in the last couple of years.
I have a confession. I have a problem knowing when to use the word "I" or "me."
"You and me are going to win," is grammatically incorrect, I know. "I" is going to win. Likewise, "In the future, clearance for lunches over $100 must be obtained from Max or I," is improper usage.
The word "I" is used when "you" is the subject of a sentence.
"I" does things. "I like that," you say. You don't say, "Me like that," unless you are Tarzan. "Me" makes an appearance when things are done to "You." "He really beat me on that deal," is correct usage.
"I" is the nominative form of the pronoun and "me" is the accusative form.
"Between you and me" is the correct form. Using "me" in the sentence is correct because it is the object of the preposition "between." Objects of a preposition should be in accusative form. Likewise, it is incorrect to say: "Me like that", and correct to say "I like that" because "I" is the nominative form and the nominative form is the correct form to use since "I" is the subject of the sentence.
We don't say give the ball to I. Thus: give the ball to John and me. If John were left out, me would stand as the correct usage.
The formula is simple: take the other out of the sentence and you will know immediately which is correct.
None of this really makes sense to me because I learned it long ago and I still make mistakes. It is easier to just rewrite the sentence so that it is not a problem. If you notice that I have used the words "me" or "I" incorrectly in the newsletter, don't tell me.
I misused it a couple of months ago and I am sorry.
Shawn:I usually decide which is right (in the more questionable cases) byseeing if I would use 'we' or 'us' when substituted. If 'we' then I use 'I', if 'us' then I use 'me'. So in your first case, I would say "we are going to win", which means I would also say "you and I are going to win". If I were to say that "the decision is up to us", I would then say that "the decision is up to you and me", similarly I would say, "Give the ball to us." For some reason, we have an easier time correctly using 'we' and 'us',even though the rule is the same. Perhaps it is because there are so many that try to improperly correct first person references to 'I', which has jumbled the synaptic connections in the language center of our brains. For example, most people would try to correct "banish you and me" to "banish you and I" although the latter is incorrect, because we all understand that they would "banish us" you know.
Especially if we were Emily Dickinson, right?
I’m nobody! Who are you?
Are you nobody, too?
Then there ’s a pair of us—don’t tell!
They ’d banish us, you know.
How dreary to be somebody!
How public, like a frog
To tell your name the livelong day
To an admiring bog!
M: Thanks, that did help. Maybe I finally understand. Love, M
Shawn: Oh yes, you pick up every subtle aspect.
This is what Uncle Ken Pitts had and what he died with. He had trouble breathing. I didn't ever know what it was and decided to look it up. Just in case you care, here it is. M What is myasthenia gravis? Myasthenia gravis is a chronic autoimmune neuromuscular disease characterized by varying degrees of weakness of the skeletal (voluntary) muscles of the body. The name myasthenia gravis, which is Latin and Greek in origin, literally means "grave muscle weakness." With current therapies, however, most cases of myasthenia gravis are not as "grave" as the name implies. In fact, for the majority of individuals with myasthenia gravis, life expectancy is not lessened by the disorder. The hallmark of myasthenia gravis is muscle weakness that increases during periods of activity and improves after periods of rest. Certain muscles such as those that control eye and eyelid movement, facial expression, chewing, talking, and swallowing are often, but not always, involved in the disorder. The muscles that control breathing and neck and limb movements may also be affected. What causes myasthenia gravis? Myasthenia gravis is caused by a defect in the transmission of nerve impulses to muscles. It occurs when normal communication between the nerve and muscle is interrupted at the neuromuscular junction - the place where nerve cells connect with the muscles they control. Normally when impulses travel down the nerve, the nerve endings release a neurotransmitter substance called acetylcholine. Acetylcholine travels through the neuromuscular junction and binds to acetylcholine receptors which are activated and generate a muscle contraction. In myasthenia gravis, antibodies block, alter, or destroy the receptors for acetylcholine at the neuromuscular junction which prevents the muscle contraction from occurring. These antibodies are produced by the body's own immune system. Thus, myasthenia gravis is an autoimmune disease because the immune system - which normally protects the body from foreign organisms - mistakenly attacks itself. What is the role of the thymus gland in myasthenia gravis? The thymus gland, which lies in the upper chest area beneath the breastbone, plays an important role in the development of the immune system in early life. Its cells form a part of the body's normal immune system. The gland is somewhat large in infants, grows gradually until puberty, and then gets smaller and is replaced by fat with age. In adults with myasthenia gravis, the thymus gland is abnormal. It contains certain clusters of immune cells indicative of lymphoid hyperplasia - a condition usually found only in the spleen and lymph nodes during an active immune response. Some individuals with myasthenia gravis develop thymomas or tumors of the thymus gland. Generally thymomas are benign, but they can become malignant. The relationship between the thymus gland and myasthenia gravis is not yet fully understood. Scientists believe the thymus gland may give incorrect instructions to developing immune cells, ultimately resulting in autoimmunity and the production of the acetylcholine receptor antibodies, thereby setting the stage for the attack on neuromuscular transmission. What are the symptoms of myasthenia gravis? Although myasthenia gravis may affect any voluntary muscle, muscles that control eye and eyelid movement, facial expression, and swallowing are most frequently affected. The onset of the disorder may be sudden. Symptoms often are not immediately recognized as myasthenia gravis. In most cases, the first noticeable symptom is weakness of the eye muscles. In others, difficulty in swallowing and slurred speech may be the first signs. The degree of muscle weakness involved in myasthenia gravis varies greatly among patients, ranging from a localized form, limited to eye muscles (ocular myasthenia), to a severe or generalized form in which many muscles - sometimes including those that control breathing - are affected. Symptoms, which vary in type and severity, may include a drooping of one or both eyelids (ptosis), blurred or double vision (diplopia) due to weakness of the muscles that control eye movements, unstable or waddling gait, weakness in arms, hands, fingers, legs, and neck, a change in facial expression, difficulty in swallowing and shortness of breath, and impaired speech (dysarthria). Who gets myasthenia gravis? Myasthenia gravis occurs in all ethnic groups and both genders. It most commonly affects young adult women (under 40) and older men (over 60), but it can occur at any age. In neonatal myasthenia, the fetus may acquire immune proteins (antibodies) from a mother affected with myasthenia gravis. Generally, cases of neonatal myasthenia gravis are transient (temporary) and the child's symptoms usually disappear within 2-3 months after birth. Other children develop myasthenia gravis indistinguishable from adults. Myasthenia gravis in juveniles is common. Myasthenia gravis is not directly inherited nor is it contagious. Occasionally, the disease may occur in more than one member of the same family. Rarely, children may show signs of congenital myasthenia or congenital myasthenic syndrome. These are not autoimmune disorders, but are caused by defective genes that produce proteins in the acetylcholine receptor or in acetylcholinesterase. How is myasthenia gravis diagnosed? Unfortunately, a delay in diagnosis of one or two years is not unusual in cases of myasthenia gravis. Because weakness is a common symptom of many other disorders, the diagnosis is often missed in people who experience mild weakness or in those individuals whose weakness is restricted to only a few muscles. The first steps of diagnosing myasthenia gravis include a review of the individual's medical history, and physical and neurological examinations. The signs a physician must look for are impairment of eye movements or muscle weakness without any changes in the individual's ability to feel things. If the doctor suspects myasthenia gravis, several tests are available to confirm the diagnosis. A special blood test can detect the presence of immune molecules or acetylcholine receptor antibodies. Most patients with myasthenia gravis have abnormally elevated levels of these antibodies. However, antibodies may not be detected in patients with only ocular forms of the disease. Another test is called the edrophonium test. This approach requires the intravenous administration of edrophonium chloride or Tensilon(r), a drug that blocks the degradation (breakdown) of acetylcholine and temporarily increases the levels of acetylcholine at the neuromuscular junction. In people with myasthenia gravis involving the eye muscles, edrophonium chloride will briefly relieve weakness. Other methods to confirm the diagnosis include a version of nerve conduction study which tests for specific muscle "fatigue" by repetitive nerve stimulation. This test records weakening muscle responses when the nerves are repetitively stimulated. Repetitive stimulation of a nerve during a nerve conduction study may demonstrate decrements of the muscle action potential due to impaired nerve-to-muscle transmission. A different test called single fiber electromyography (EMG), in which single muscle fibers are stimulated by electrical impulses, can also detect impaired nerve-to-muscle transmission. EMG measures the electrical potential of muscle cells. Muscle fibers in myasthenia gravis, as well as other neuromuscular disorders, do not respond as well to repeated electrical stimulation compared to muscles from normal individuals. Computed tomography (CT) may be used to identify an abnormal thymus gland or the presence of a thymoma. A special examination called pulmonary function testing - which measures breathing strength - helps to predict whether respiration may fail and lead to a myasthenic crisis. How is myasthenia gravis treated? Today, myasthenia gravis can be controlled. There are several therapies available to help reduce and improve muscle weakness. Medications used to treat the disorder include anticholinesterase agents such as neostigmine and pyridostigmine, which help improve neuromuscular transmission and increase muscle strength. Immunosuppressive drugs such as prednisone, cyclosporine, and azathioprine may also be used. These medications improve muscle strength by suppressing the production of abnormal antibodies. They must be used with careful medical followup because they may cause major side effects. Thymectomy, the surgical removal of the thymus gland (which often is abnormal in myasthenia gravis patients), reduces symptoms in more than 70 percent of patients without thymoma and may cure some individuals, possibly by re-balancing the immune system. Other therapies used to treat myasthenia gravis include plasmapheresis, a procedure in which abnormal antibodies are removed from the blood, and high-dose intravenous immune globulin, which temporarily modifies the immune system and provides the body with normal antibodies from donated blood. These therapies may be used to help individuals during especially difficult periods of weakness. A neurologist will determine which treatment option is best for each individual depending on the severity of the weakness, which muscles are affected, and the individual's age and other associated medical problems. What are myasthenic crises? A myasthenic crisis occurs when the muscles that control breathing weaken to the point that ventilation is inadequate, creating a medical emergency and requiring a respirator for assisted ventilation. In patients whose respiratory muscles are weak, crises - which generally call for immediate medical attention - may be triggered by infection, fever, or an adverse reaction to medication. What is the prognosis? With treatment, the outlook for most patients with myasthenia gravis is bright: they will have significant improvement of their muscle weakness and they can expect to lead normal or nearly normal lives. Some cases of myasthenia gravis may go into remission temporarily and muscle weakness may disappear completely so that medications can be discontinued. Stable, long-lasting complete remissions are the goal of thymectomy. In a few cases, the severe weakness of myasthenia gravis may cause a crisis (respiratory failure), which requires immediate emergency medical care. What research is being done? Within the Federal Government, the National Institute of Neurological Disorders and Stroke (NINDS), one of the Federal Government's National Institutes of Health (NIH), has primary responsibility for conducting and supporting research on myasthenia gravis. Much has been learned about myasthenia gravis in recent years. Technological advances have led to more timely and accurate diagnosis, and new and enhanced therapies have improved management of the disorder. Much knowledge has been gained about the structure and function of the neuromuscular junction, the fundamental aspects of the thymus gland and of autoimmunity, and the disorder itself. Despite these advances, however, there is still much to learn. The ultimate goal of myasthenia gravis research is to increase scientific understanding of the disorder. Researchers are seeking to learn what causes the autoimmune response in myasthenia gravis, and to better define the relationship between the thymus gland and myasthenia gravis. Today's myasthenia gravis research includes a broad spectrum of studies conducted and supported by NINDS. NINDS scientists are evaluating new and improving current treatments for the disorder. One such study is testing the efficacy of intravenous immune globlin in patients with myasthenia gravis. The goal of the study is to determine whether this treatment safely improves muscle strength. Another study seeks further understanding of the molecular basis of synaptic transmission in the nervous system. The objective of this study is to expand current knowledge of the function of receptors and to apply this knowledge to the treatment of myasthenia gravis. Where can I get more information? For more information on neurological disorders or research programs funded by the National Institute of Neurological Disorders and Stroke, contact the Institute's Brain Resources and Information Network (BRAIN) at: BRAIN P.O. Box 5801 Bethesda, MD 20824 (800) 352-9424 http://www.ninds.nih.gov Information also is available from the following organizations: Myasthenia Gravis Foundation of America, Inc. 1821 University Ave W. Suite S256 St. Paul, MN 55104-2897 mgfa@myasthenia.org http://www.myasthenia.org Tel: 800-541-5454 651-917-6256 Fax: 651-917-1835 Muscular Dystrophy Association 3300 East Sunrise Drive Tucson, AZ 85718-3208 mda@mdausa.org http://www.mda.org Tel: 520-529-2000 800-344-4863 Fax: 520-529-5300 "Myasthenia Gravis Fact Sheet," NINDS. NIH Publication No. 99-768 Prepared by: Office of Communications and Public Liaison National Institute of Neurological Disorders and Stroke National Institutes of Health Bethesda, MD 20892 NINDS health-related material is provided for information purposes only and does not necessarily represent endorsement by or an official position of the National Institute of Neurological Disorders and Stroke or any other Federal agency. Advice on the treatment or care of an individual patient should be obtained through consultation with a physician who has examined that patient or is familiar with that patient's medical history. All NINDS-prepared information is in the public domain and may be freely copied. Credit to the NINDS or the NIH is appreciated. Last updated February 14, 2007
Edward Everett Hale: I am only one, But still I am one. I cannot do everything, But still I can do something; And because I cannot do everything I will not refuse to do the something that I can do.
And a time for every matter under heaven:
A time to be born, and a time to die;
A time to plant, and a time to pluck up what is planted;
A time to kill, and a time to heal;
A time to break down, and a time to build up;
A time to weep, and a time to laugh;
A time to mourn, and a time to dance;
A time to throw away stones, and a time to gather stones together;
A time to embrace, And a time to refrain from embracing;
A time to seek, and a time to lose;
A time to keep, and a time to throw away;
A time to tear, and a time to sew;
A time to keep silence, and a time to speak;
A time to love, and a time to hate,
A time for war, and a time for peace.
Ecclesiastes 3:1-8
A time for the family newsletter.
Mona wrote: So sorry to read about Melanie and we Pray things are not all so big and hard. We can and will Pray. Love Mona and Von Grundvig
Ella Dean Hunter wrote: I'm sorry to hear about Melanie. I assume this is your daughter. Please keep me posted.I talked with Joyce W this afternoon. It was nice to talk to her. They were on their way to Vernal to a reunion.Ella DeanCharlotte wrote: I just read the email and am concerned about Melanie. What is going on? Love CharMyrna wrote: We still don't know. It seems that she MAY have MS. She has further tests on Wednesday. It is likely either a tumor or MS. Love, MHelen wrote: Just reading my e-mails. Thank you for keeping me informed. Love to both of you. Helen
This summer has really flown by and I can't believe that school will be starting in less than a week on the 22nd. Tyler and Emily want summer to last longer, but Michael is ready for school to begin this year. I know, I even checked his temperature after I heard him! This will be a short one this month since we haven't been doing many new things to tell you all about. Tyler and Emily's soccer season is up and running and this year they are on the same team (yay!). Which means half of the running around than last year. All 3 kids are now back in piano and are getting their pieces put together for the Pianorama at the end of October. Pianorama is a piano ensemble event that is held every 2 years in central Ohio. Each student is given half (Primo or Secondo) of 3 duet pieces to learn and then will be paired with another student to perform their whole piece. On the day of the Pianorama performance there will be 22 pianos on one stage with 44 students playing the same songs all together. There are 9 different playing levels that will perform so Michael, Tyler and Emily are all in different groups. I hear it is quite amazing to see and hear and their teacher tells me that it's pretty hard to get in, so we are lucky to have this opportunity. Todd is staying busy with his 2 callings in the ward. Scoutmaster over 2 wards and 2nd counselor in the YM presidency. We (the whole family) just went on a combined youth outing this week with Todd and Michael where we went hiking in a place called Hocking Hills State Park. It was so beautiful and reminded us of Zions Canyon. Hailey absolutely loved it and insisted on hiking everywhere herself and NOT be carried. It made for some slow walking at times, but it was fun to watch her be so independent. Any time anyone wants to come and visit us during warm weather, we would love to take you there. Our kids want to go again, soon! We really enjoyed seeing everyone this July and feel bad that our last 9 days of Utah were kind of a waste with having sick kids and not going anywhere or really seeing anyone for fear of spreading the muck. We love you all and just know that our home is open anytime to any and all who would like to come and see the Buckeye State! Love to all, Todd, Amy, Michael, Tyler, Emily and Hailey
Yep, I know that is what you are going to say: that darn newsletter! Anyway, no matter what you say or do, I will keep them coming. Remember, I am good at making up things. Are lies better than truth? Unless you want to find out--WRITE! Love, M
Shawn wrote: Are lies better than the truth? Well, even though a lie could free us from writing for the newsletter, we all really know that only the truth can make us free.
Dear M, Thank you so much for "The Reluctant Gift: Putting Our Time on the Altar." Recently I have had a special opportunity to be available much more often to care for two of my grandchildren -- 2-year old and a 5-month old. It truly is a blessing to me. The bonds of love grow stronger when we serve and make ourselves available to serve. I appreciate so much the article, The Reluctant Gift. Actually, Myrna, I know that you are one who always has and always will go the extra miles to serve and help others. Thanks so much for the great example you and Leonard have been in my life! Take Care, Ann
HUNTINGTON, Utah (AP) - A coal mine collapsed Monday in central Utah, trapping six miners less than 20 miles from the epicenter of a 4.0 magnitude earthquake, authorities said.
The Genwal mine reported a "cave-in" at 3:50 a.m. MDT, an hour after the earthquake, the Emery County sheriff's office said.
"Rescue workers are on scene trying to locate six miners that are unaccounted for," the sheriff's office. Rocky Mountain Power, a utility with a power plant in the area, sent a rescue team and heavy equipment, spokesman Dave Eskelsen said.
A command center was being set up in Huntington, about 15 miles from the mine, said Teresa Behunin, an accountant affiliated with the mine. She had no other details.
Todd wrote: Not anyone we know, I hope?
Myrna wrote: I don't know. They are having a difficult time getting to them. Dad and I tried to call Garth but he and Alyce were in Provo getting his treatment so we left a message and they have not called back. Love, M
On Jul 29, 2007, at 3:54 PM, Eric trauntvein wrote:
A guy was talking to me at work telling me how dangerous my soda was, I told him his diet was worse because the body treats Aspartame the same as sugar. I was wrong! the following attachments I am sending will tell you why I was wrong. there are just a few but there is a lot of solid proof send it around . Yes, I was wrong about the body treating Aspartame the same as sugar, how can one confuse poison with sugar, love eric
Got a little couch potato? Check out fun summer activities for kids.
David wrote: Your first statement was right in some regards. I heard this on the radio asone of the latest medical discoveries.
http://www.webmd.com/diet/news/20050613/drink-more-diet-soda-gain-more-weigh
Eric wrote: I just wish that, someday, something I decide to do will be right. Sugar became the enemy in the '80s and now aspartame is the bad guy. I should have stayed with water.Aspartame, a high intensity sweetener marketed under brands such as Equal, NutraSweet and Canderel, was first discovered in 1965. But because of initial cancer concerns, it was not approved for use in dry goods until the early 1980s. Over the next score of years, its use slowly expanded to beverages and other food products, until all restrictions for its use were removed by the US Food and Drug Administration (FDA) in 1996.Although industry now keenly points to over 200 studies that prove its safety, aspartame has not fully rid itself of the black spot tainting its image.Two years after one of the world's few comprehensive cancer-testing programs, the European Ramazzini Foundation (ERF), published its first study linking aspartame to cancer, the foundation last month (July 2007) again flagged up the dreaded link. And this second study has - again - resulted in a whirlwind of comment and controversy.The Journal of the American Medical Association suggests that there is a link between sugar sweetened sodas and diabetes. Analysis of the eight year Nurses' Health Study II shows that women who increased their sugar sweetened soft drink consumption from one soda per week to one or more sugar sweetened soft drinks per day gained weight and had a higher risk of type II diabetes. Women who decreased their consumption of sugar sweetened sodas tended to lose weight and had a lower frequency of type II diabetes.The best solution is to drink water. Drink at least half of your body weight in ounces of pure water every day.